681 research outputs found
Geometric Approach to Lyapunov Analysis in Hamiltonian Dynamics
As is widely recognized in Lyapunov analysis, linearized Hamilton's equations
of motion have two marginal directions for which the Lyapunov exponents vanish.
Those directions are the tangent one to a Hamiltonian flow and the gradient one
of the Hamiltonian function. To separate out these two directions and to apply
Lyapunov analysis effectively in directions for which Lyapunov exponents are
not trivial, a geometric method is proposed for natural Hamiltonian systems, in
particular. In this geometric method, Hamiltonian flows of a natural
Hamiltonian system are regarded as geodesic flows on the cotangent bundle of a
Riemannian manifold with a suitable metric. Stability/instability of the
geodesic flows is then analyzed by linearized equations of motion which are
related to the Jacobi equations on the Riemannian manifold. On some geometric
setting on the cotangent bundle, it is shown that along a geodesic flow in
question, there exist Lyapunov vectors such that two of them are in the two
marginal directions and the others orthogonal to the marginal directions. It is
also pointed out that Lyapunov vectors with such properties can not be obtained
in general by the usual method which uses linearized Hamilton's equations of
motion. Furthermore, it is observed from numerical calculation for a model
system that Lyapunov exponents calculated in both methods, geometric and usual,
coincide with each other, independently of the choice of the methods.Comment: 22 pages, 14 figures, REVTeX
FIA functions as an early signal component of abscisic acid signal cascade in Vicia faba guard cells
An abscisic acid (ABA)-insensitive Vicia faba mutant, fia (fava bean impaired in ABA-induced stomatal closure) had previously been isolated. In this study, it was investigated how FIA functions in ABA signalling in guard cells of Vicia faba. Unlike ABA, methyl jasmonate (MeJA), H2O2, and nitric oxide (NO) induced stomatal closure in the fia mutant. ABA did not induce production of either reactive oxygen species or NO in the mutant. Moreover, ABA did not suppress inward-rectifying K+ (Kin) currents or activate ABA-activated protein kinase (AAPK) in mutant guard cells. These results suggest that FIA functions as an early signal component upstream of AAPK activation in ABA signalling but does not function in MeJA signalling in guard cells of Vicia faba
Search for the decay
We performed a search for the decay with the
E391a detector at KEK. In the data accumulated in 2005, no event was observed
in the signal region. Based on the assumption of
proceeding via parity-violation, we obtained the single event sensitivity to be
, and set an upper limit on the branching ratio to
be at the 90% confidence level. This is a factor of 3.2
improvement compared to the previous results. The results of proceeding via parity-conservation were also presented in this paper
Long-lived neutral-kaon flux measurement for the KOTO experiment
The KOTO ( at Tokai) experiment aims to observe the CP-violating rare
decay by using a long-lived neutral-kaon
beam produced by the 30 GeV proton beam at the Japan Proton Accelerator
Research Complex. The flux is an essential parameter for the measurement
of the branching fraction. Three neutral decay modes, , , and were used to
measure the flux in the beam line in the 2013 KOTO engineering run. A
Monte Carlo simulation was used to estimate the detector acceptance for these
decays. Agreement was found between the simulation model and the experimental
data, and the remaining systematic uncertainty was estimated at the 1.4\%
level. The flux was measured as per protons on a
66-mm-long Au target.Comment: 27 pages, 16 figures. To be appeared in Progress of Theoretical and
Experimental Physic
Experimental study of the decay
The first dedicated search for the rare neutral-kaon decay
has been carried out in the E391a experiment at the
KEK 12-GeV proton synchrotron. The final upper limit of 2.6 at
the 90% confidence level was set on the branching ratio for the decay.Comment: 23 pages, 27 figures, accepted for publication as a regular article
in Physical Review
Human PSF binds to RAD51 and modulates its homologous-pairing and strand-exchange activities
RAD51, a eukaryotic recombinase, catalyzes homologous-pairing and strand-exchange reactions, which are essential steps in homologous recombination and recombinational repair of double strand breaks. On the other hand, human PSF was originally identified as a component of spliceosomes, and its multiple functions in RNA processing, transcription and DNA recombination were subsequently revealed. In the present study, we found that PSF directly interacted with RAD51. PSF significantly enhanced RAD51-mediated homologous pairing and strand exchange at low RAD51 concentrations; however, in contrast, it inhibited these RAD51-mediated recombination reactions at the optimal RAD51 concentration. Deletion analyses revealed that the N-terminal region of PSF possessed the RAD51- and DNA-binding activities, but the central region containing the RNA-recognition motifs bound neither RAD51 nor DNA. These results suggest that PSF may have dual functions in homologous recombination and RNA processing through its N-terminal and central regions, respectively
Search for short baseline nu(e) disappearance with the T2K near detector
8 pages, 6 figures, submitted to PRD rapid communication8 pages, 6 figures, submitted to PRD rapid communicationWe thank the J-PARC staff for superb accelerator performance and the CERN NA61 collaboration for providing valuable particle production data. We acknowledge the support of MEXT, Japan; NSERC, NRC and CFI, Canada; Commissariat `a l’Energie Atomique and Centre National de la Recherche Scientifique–Institut National de Physique Nucle´aire et de Physique des Particules, France; DFG, Germany; INFN, Italy; National Science Centre (NCN), Poland; Russian Science Foundation, RFBR and Ministry of Education and Science, Russia; MINECO and European Regional Development Fund, Spain; Swiss National Science Foundation and State Secretariat for Education, Research and Innovation, Switzerland; STFC, UK; and DOE, USA. We also thank CERN for the UA1/NOMAD magnet, DESY for the HERA-B magnet mover system, NII for SINET4, the WestGrid and SciNet consortia in Compute Canada, GridPP, UK. In addition participation of individual researchers and institutions has been further supported by funds from ERC (FP7), EU; JSPS, Japan; Royal Society, UK; DOE Early Career program, USA
Measurement of the CP Violation Parameter sin(2phi_1) in B^0_d Meson Decays
We present a measurement of the Standard Model CP violation parameter
sin(2phi_1) based on a 10.5 fb^{-1} data sample collected at the Upsilon(4S)
resonance with the Belle detector at the KEKB asymmetric e+e- collider. One
neutral B meson is reconstructed in the J/psi K_S, psi(2S) K_S, chi_{c1} K_S,
eta_c K_S, J/psi K_L or J/psi pi^0 CP-eigenstate decay channel and the flavor
of the accompanying B meson is identified from its charged particle decay
products. From the asymmetry in the distribution of the time interval between
the two B-meson decay points, we determine sin(2phi_1) = 0.58 +0.32-0.34 (stat)
+0.09-0.10 (syst).Comment: LaTex, 13 pages, 3 figures, submitted to P.R.
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